We have been heartened by the increased mainstream media coverage of healthy hearing lately.

The road to more effective, less invasive, and faster developing treatments for tinnitus and loudness hyperacusis lies in focusing on the brain and not the ear.

Let’s push back against the idea of a “loss” or “impairment” when it comes to hearing.

This suggests that birds maintain a precise program for hair cell regeneration that preserves frequency-specific nerve connections, which is an important aspect of proper functional recovery.

Our son with a hearing loss opened up our family’s world and made us all stronger.

Perhaps most of all, the message of enjoying responsibly is one we really try to impart.

Of relevance to hyperacusis, prior noise-induced hearing loss leads to the generation of prolonged and repetitive activity in type II neurons and surrounding tissues. This aberrant signaling may be the basis for the sensitivity to everyday sounds seen in hyperacusis.

Sometimes, communication snafus can be comedy gold.

These findings suggest that the auditory cortex may transmit significant non-auditory signals relevant for learning-related plasticity.

“Lacking social connection is as dangerous as smoking up to 15 cigarettes a day,” according to a U.S. Surgeon General report.

As a result, increased prefrontal activity may be helpful in improving gating function (automatic filtering), a topic for future research.

Our independent analysis of the clinical trials’ published data yields the following insights on gene therapy for hearing loss from otoferlin variants. The clinical trials provide the first validation of pharmacological treatments for hearing loss.